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Inflammation of the kidney tubules and interstitium — most often drug-induced acute interstitial nephritis.
Acute interstitial nephritis (AIN) is commonly caused by NSAIDs, PPIs, and antibiotics, presenting with rising creatinine weeks after exposure, sometimes with fever and eosinophilia. Removing the culprit usually recovers kidney function; steroids are used in severe cases. Chronic interstitial nephritis follows long-term exposure (analgesics, lithium, heavy metals).
A sudden drop in kidney function over hours to days - reversible if caught early, but a major risk factor for chronic kidney disease.
Kidney injury caused by NSAIDs (ibuprofen, diclofenac, naproxen) — a leading preventable cause of AKI and CKD progression.
Abnormal presence of protein in the urine, a key marker of kidney damage and the strongest predictor of CKD progression and cardiovascular risk.
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This content is a general reference, not medical advice, a diagnosis, or a treatment plan. Do not change your diet, fluids, medicines, or dialysis plan without your nephrologist or renal dietitian. Individual recommendations depend on your labs, medications, conditions, and care plan.